A Hidden Secondary Hyperparathyroidism: A Case Report
M. A. Amani1
M. J. Yousfi2
1 Department of Endocrinology, 1st November 1954 University Hospital of Oran, Algeria
2 Department of Urology, 1st November 1954 University Hospital of Oran, Algeria
International Journal of Clinical Case Reports, 2016, Vol. 6, No. 31 doi: 10.5376/ijccr.2016.06.0031
Received: 06 Nov., 2016 Accepted: 12 Dec., 2016 Published: 13 Dec., 2016
© 2016 BioPublisher Publishing Platform
This is an open access article published under the terms of the Creative Commons Attribution License
, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Preferred citation for this article:
Amani M.A., Miraoui S. and Yousfi M.J., 2016, A Hidden Secondary Hyperparathyroidism: A Case Report, International Journal of Clinical Case Reports, 6(31): 1-2 (doi: 10.5376/ijccr.2016.06.0031)
The secondary hyperparathyroidism is defined by an elevated parathyroid hormone (PTH) secondary to a decrease in calcemia. We report the case of a 15-year old female, followed for 3 years at the nephrology department for a nephrocalcinosis and which was addressed to us for the suspicion of a primary hyperparathyroidism. We noted in her personal history renal lithiasis.
The physical examination found a tooth dystrophy with decalcified teeth and diffuse bone pain. First blood tests: calcemia: 85mg/L (80-105), phosphoremia: 55mg/L (40-70) and PTH: 147.6pg/ml high (15-65). Second blood tests: calcemia: 90.85mg/L (81-104), phosphoremia: 40.14mg/L (25-46) and PTH: 169.4pg/ml high (15-65). The blood tests were completed with a dosage of 25-hydroxy-vitamin D: 11ng/ml (deficiency), 24 hours calciuria: 198mg/24h (4.4mg/Kg/24h), high. The renal ultrasound showed a nephrocalcinosis. The patient received a vitamin D treatment than the normalizing of the vitamin D was obtained (30.53ng/ml) and PTH too (56pg/ml, standards: 15-80). The diagnosis of a primary hyperparathyroidism will be retained after eliminating a secondary hyperparathyroidism.
Secondary hyperparathyroidism; Hypercalcemia; Vitamin D; Nephrocalcinosis